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The objectives of our research are to obtain information regarding mechanisms of trophic control in the avian retina that can be applied to mammalian retinae. The avian retina is ideal for this study because of the animal’s unique ability to regulate its electroretinogram (ERG) amplitude via changes in retinal pigment epithelium (RPE) transport. The visual streak of the avian retina, like the mammalian fovea, concentrates the density of rods and cones that are concentrated in the visual streak. We will therefore study the control of ganglion cell function by the cells that supply the RPE in the avian retina. We propose to study the anatomic and physiological circuitry that links the cells responsible for the RPE-mediated regulation of ERG amplitude, the RPE cells and the photoreceptors, and the role of the optic nerve in this control. In the initial studies, we will determine the anatomical and physiological nature of the feedback control. Also, we will study the role of the optic nerve in the control process. Anatomical studies will include labeling the cells of the retina with intracellular recording and stimulating electrodes from the optic nerve to determine the nature of the electrical feedback signal and the type of optic nerve cells (retinal ganglion cells, optic nerve cells) that receive it. Physiological studies will involve the determination of the amplitude of the feedback signal, its frequency, the number of optic nerve cells that receive it, and the minimum amplitude that produces an effect. In the final set of experiments, we will investigate the mechanism of the regulation, by studying the effect of surgical optic nerve section on the amplitude of the feedback signal. Results of these studies should be of immediate relevance to understanding the control of retinal ganglion cell function in the visual system of higher mammals.include(CheckIncludeFile)

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